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. 2019 Oct 17;7:e7815. doi: 10.7717/peerj.7815

Figure 6. VEGF inhibitor avastin prevented the role of RSG in BM-EPCs induced by high glucose.

Figure 6

(A) BM-EPCs exposed to 33 mmol/L glucose with or without RSG (10 nM) and VEGF inhibitor Avastin (25 µg/ml) for 24 h. CCK-8 detected EPC viability in vitro. (B–F) Avastin abolished the elevated tube formation ability by RSG (C–F: 50×; scale bar, 100µm). Values are mean ± SEM, (n = 6 per group). RSG, rosiglitazone. HG, high glucose.