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. Author manuscript; available in PMC: 2020 Nov 1.
Published in final edited form as: Acta Neuropathol. 2019 Aug 7;138(5):729–749. doi: 10.1007/s00401-019-02054-4

Fig. 1.

Fig. 1.

Pathologic NFT inclusions and amyloid-β plaques accumulate in vulnerable brain regions during Alzheimer’s disease. Sections of mid hippocampus were a stained with hematoxylin and eosin plus luxol fast blue (H&E/LFB) or b used for immunohistochemical staining with PHF-tau antibody. Sections of the superior temporal gyrus were c stained with H&E/LFB or d used for immunohistochemical staining with amyloid-β antibody. Neurofibrillary degeneration and scattered neuritic plaques are visualized at × 200 (e) and at × 400 (f) in the hippocampal section incubated with PHF-tau antibody. Diffuse and dense core plaques are visualized at × 200 (g) and at × 400 (h) in the superior temporal gyrus section incubated with amyloid-β Antibody