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. 2019 Sep 10;11(3):24. doi: 10.1038/s41368-019-0055-0

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — The expression of p-MAPKs in vivo after vehicle administration and 24 h after CFA injection. a–c IF staining of p-MAPKs in rat TG slices in the vehicle and CFA groups. Arrows (solid) show that p-MAPKs co-localized with GS, and arrows (hollow) show that p-MAPKs localized in TG neurons (green: GS; red: p-ERK1/2, p-P38, and p-JNK; blue: DAPI). Scale bars: 50 μm. WB images and graphs in d–f show that the expression of p-ERK1/2, p-P38 and p-JNK in CFA-treated TGs is higher than that in vehicle TGs, and these levels were normalized against their total levels. (n = 6, *P < 0.05 compared with the vehicle group)