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. 2019 Jun 19;9(5):1035–1049. doi: 10.1016/j.apsb.2019.06.003

Table 4.

Desirability of antibiotic‒XueBiJing compound pairs.

Antibiotic‒XueBiJing pair Interacting protein Inhibition DDI index Potential outcome Remark
Cefoperazone (A8)‒senkyunolide I (X5) UGT2B15 0.12 Decreased glucuronidation of and increased systemic exposure to X5. For X5, its unbound Cmax 0.2 μmol/L (Table 1) is less than its effective concentration 10 μmol/L (Supporting Information Table S2), so inhibition of UGT2B15 by these antibiotics at the clinically relevant doses would lead to the desirable concentration. Probably desirable due to the low baseline level of systemic exposure to X5.
Penicillin G (A15)‒senkyunolide I (X5) 0.43–0.74
Piperacillin (A13)‒senkyunolide I (X5) 0.56
Voriconazole (A45)‒senkyunolide I (X5) 0.75
Cefoxitin (A20)‒senkyunolide I (X5) 1.28
Oxacillin (A17)‒protocatechuic aldehyde ALDH 0.11 Decreased systemic exposure to the metabolite X11 and increased methylated protocatechuic aldehyde. For X11, its unbound Cmax 0.03 μmol/L (Table 1) is low as compared with its effective concentrations 1–130 μmol/L (Supporting Information Table S2); inhibition of ALDH by these antibiotics at the clinically relevant doses would further decrease its concentration. Undesirable but probably of limited therapeutic relevance due to the low baseline level of systemic exposure to X11.
Ampicillin (A16)‒protocatechuic aldehyde 0.17
Ceftazidime (A7)‒protocatechuic aldehyde 0.19
Flucloxacillin (A19)‒protocatechuic aldehyde 0.62
Meropenem (A2)‒protocatechuic aldehyde 0.78
Penicillin G (A15)‒protocatechuic aldehyde 1.24–2.14
Imipenem (A1)‒protocatechuic aldehyde 2.14
Flucloxacillin (A19)‒tanshinol (X8) OAT1 0.16 Decreased renal excretion of and increased systemic exposure to X8. For X8, its unbound Cmax 0.1 μmol/L (Table 1) is less than its effective concentrations 1–25 μmol/L (Supporting Information Table S2), so inhibition of OAT1/2 by these antibiotics at the clinically relevant doses would lead to the desirable concentrations. Probably desirable due to the low baseline level of systemic exposure to X8.
Voriconazole (A45)‒tanshinol (X8) 0.22
Ticarcillin (A14)‒tanshinol (X8) 0.26
Cefamandole (A11)‒tanshinol (X8) 0.33
Penicillin G (A15)‒tanshinol (X8) 0.38–0.66
Cefoperazone (A8)‒tanshinol (X8) 0.95–2.44
Carbenicillin (A18)‒tanshinol (X8) 1.31
Levofloxacin (A31)‒tanshinol (X8) OAT2 0.36
Oxacillin (A17)‒tanshinol (X8) OAT1/2 0.52/0.11
Piperacillin (A13)‒tanshinol (X8) 0.75/0.37
Ciprofloxacin (A32)‒tanshinol (X8) 1.96/0.55
Trimethoprim (A37)‒salvianolic acid B (X10) OATP1B3 0.12 Decreased hepatobiliary excretion of and increased systemic exposure to X10. For X10, its unbound Cmax 0.003 μmol/L (Table 1) is less than its effective concentrations 0.1–1.4 μmol/L (Supporting Information Table S2), so inhibition of OATP1B3 by these antibiotics at the clinically relevant doses would lead to the desirable concentrations. Probably desirable due to the low baseline level of systemic exposure to X10.
Amphotericin B (A42)‒salvianolic acid B (X10) 0.22
Moxifloxacin (A33)‒salvianolic acid B (X10) 0.29–0.41
Clindamycin (A35)‒salvianolic acid B (X10) 0.30
Caspofungin (A41)‒salvianolic acid B (X10) 0.35
Erythromycin (A34)‒salvianolic acid B (X10) 0.35–1.04
Teicoplanin (A26)‒salvianolic acid B (X10) 0.60
Ceftriaxone (A6)‒salvianolic acid B (X10) 0.68
Ciprofloxacin (A32)‒salvianolic acid B (X10) 0.69
Flucloxacillin (A19)‒salvianolic acid B (X10) 1.07
Cefamandole (A11)‒salvianolic acid B (X10) 1.70
Cefoperazone (A8)‒salvianolic acid B (X10) 1.72–4.43
Cefotaxime (A9)‒salvianolic acid B (X10) 1.81–2.14
Voriconazole (A45)‒salvianolic acid B (X10) 2.16
Rifamcin (A39)‒salvianolic acid B (X10) 2.63
Cefoxitin (A20)‒salvianolic acid B (X10) 2.81
Oxacillin (A17)‒salvianolic acid B (X10) 4.64
Penicillin G (A15)‒salvianolic acid B (X10) 6.8–11.8
Ampicillin (A16)‒salvianolic acid B (X10) 8.55