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. 2019 Oct 17;10:2394. doi: 10.3389/fimmu.2019.02394

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Copyright © 2019 Li, Xue, Fu, Yin, Feng and Liu.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Graphical representation showing the fold change analysis of different gene subgroups [“classical antiviral IFN-stimulated genes (ISGs),” “weakly IFN-λ3-induced genes,” and “strongly IFN-λ3-induced genes”]. (A) The classical antiviral IFN-stimulated gene (ISG) subgroup. “Classical ISGs” were defined by their designation in the literature. (B) The weakly IFN-λ3-induced gene subgroup. “Weakly IFN-λ3-induced genes:” fold increase by IFN-λ3 over mock control/fold increase by IFN-α over mock control >2 and fold increase by IFN-α over mock control >4. Unknown genes were named as UN1, UN2, and UN3. (C) The strongly IFN-λ3-induced gene subgroup. “Strongly IFN-λ3-induced genes:” fold increase by IFN-λ3 over mock control/fold increase by IFN-α over mock control >4 and fold increase by IFN-α over mock control <2.