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. 2019 Oct 4;11(19):8139–8155. doi: 10.18632/aging.102309

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Copyright © 2019 Ding et al.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY 3.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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Restored expression of MEG3 inhibits the development of gastric cancer cells. (A) The number of clones was significantly reduced in the lncRNA MEG3-overexpressing group and the 5-Aza group compared with the NC group. *P<0.05, **P<0.01, compared with the NC group. #P<0.05, compared with the 5-Aza group. (B) The apoptosis rate of MGC-803 cells transfected with MEG3 or 5-Aza dramatically increased compared with the NC group. *P<0.05, **P<0.01, compared with the NC group. #P<0.05, compared with the 5-Aza group. (C–D) Wound healing and transwell assays showed significantly reduced cell migration and invasion ability in the MEG3 and 5-Aza groups compared to the control group. *P<0.05, **P<0.01, compared with the NC group. #P<0.05, compared with the 5-Aza group.