Figure 4.

Sodium hydrosulphide (NaHS) reversed the effect of tumour necrosis factor (TNF)‐α on peroxisome proliferator‐activated receptors (PPARs). Representative immunoblots and average data of cardiac PPAR‐α, PAPR‐γ and PPAR‐δ protein levels from cells treated with TNF‐α (25 ng/mL), or NaHS (0.1 mmol/L) combined with TNF‐α for 24 h. Densitometry was normalized to glyceraldehyde 3‐phosphate dehydrogenase (GAPDH) as an internal control. Data are shown as mean ± SEM from four independent experiments