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Model of adipogenesis action of ERβ is mediated by a negative crosstalk with PPARγ. During early healing process of tendon injury, absence of ERβ will augment the PPARγ expression because of the negative crosstalk between the two so that PPARγ signalling is activated including upstream CD36 and target FABP4, the result is more TDSCs differentiating into adipocytes leads to a poor repair
