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. 2019 Jun 19;56(9):607–616. doi: 10.1136/jmedgenet-2018-105965

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© Author(s) (or their employer(s)) 2019. Re-use permitted under CC BY-NC. No commercial re-use. See rights and permissions. Published by BMJ.

This is an open access article distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited, appropriate credit is given, any changes made indicated, and the use is non-commercial. See: http://creativecommons.org/licenses/by-nc/4.0/.

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SNP effects on soluble adhesion molecule levels versus other cardiovascular health-related traits in the ABO locus. The Pearson’s r of the genetic effects (Z-scores) were estimated using a set of SNPs that located in the ABO locus (defined as the LD block²⁵ containing ABO gene in the gwas-pw²¹ analyses) and that were available in both the present study and open-access data sets.^22–24 Positive correlations are indicated with red color, negative correlations are indicated with blue color, and correlations with p≥0.05 are left blank. The scatter plot representations as well as correlations in the other loci are shown in online supplementary figure S3. CAD, coronary artery disease; HDL-C, high-density lipoprotein cholesterol; LD, linkage disequilibrium; LDL-C, low-density lipoprotein cholesterol; sE-selectin; soluble E-selectin; sICAM-1, soluble intercellular adhesion molecule-1; sVCAM-1, soluble vascular cell adhesion molecule-1; TG, total triglycerides; TotC, total cholesterol.