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. 2019 May 27;170(2):549–561. doi: 10.1093/toxsci/kfz122

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Published by Oxford University Press on behalf of the Society of Toxicology 2019.

This work is written by US Government employees and is in the public domain in the US.

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Figure 7. — Hepatic inflammation and oxidative stress in APAP-treated mice was increased by TGFβ1 signaling. A, IL-1β protein concentration as measured by ELISA assay in liver homogenates from vehicle and 6 h APAP-treated mice injected with DMSO or GW788388. B, TNFα protein concentration as measured by ELISA assay in liver homogenates from vehicle and 6 h APAP-treated mice injected with DMSO or GW788388. C, SOD1 immunoblot in liver homogenates in vehicle and 6 h APAP-treated mice administered DMSO or GW788388. D, Quantification of SOD1 immunoblots in livers of vehicle and 6 h APAP-treated mice injected with DMSO or GW788388. *p < .05 compared with vehicle-treated mice injected with DMSO. #p < .05 compared with 6 h APAP-treated mice injected with DMSO.