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. 2019 Oct 31;10:4970. doi: 10.1038/s41467-019-12956-2

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — Chemical–genetic interactions. a Summary of chemical–genetic interactions. Red lines represent direct interactions that are consistent (solid red lines) or inconsistent (dotted red lines) with the chemical–genetic interactions defined in this study. Brown lines represent chemical–genetic interactions that have been defined in this study and are most likely indirect. Impact of piericidin A (b, h) bedaquiline (c) thioridazine (d), clofazimine (e), DDD00853663 (f), and Q203 (g) on growth. Data are averages of three cultures and represent at least two independent experiments. Error bars correspond to standard deviation. BDQ, bedaquiline; CFZ, clofazimine; PHT, phenothiazines; PIR, piericidin A; PYR, pyridaben; ROT, rotenone; 2-MQ, 2-mercapto-quinazolinones. “Comp” stands for complemented. Source data are provided as a Source Data file