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. 2018 Apr 9;43(4):550–561. doi: 10.1016/j.jgr.2018.04.001

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© 2018 The Korean Society of Ginseng, Published by Elsevier Korea LLC.

This is an open access article under the CC BY-NC-ND license (http://creativecommons.org/licenses/by-nc-nd/4.0/).

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Fig. 9 — Effects of 20 (S)-ginsenoside Rg3 on EGFR expressions. (A) Immunohistochemical analyses of EGFR in different groups in the alcohol GU model, pylorus-ligated GU model, and acetic acid GU model (×200). Positive NOS2 staining cells of different groups in the (B) alcohol GU model, (C) pylorus-ligated GU model, and (D) acetic acid GU model. Data are presented as the mean ± SD (n = 10; compared with the blank control group, ^#p < 0.05, ^##p < 0.01, ^###p < 0.001; compared with the model control group, *p < 0.05, **p < 0.01, ***p < 0.001).

EGFR, epidermal growth factor receptor; GU, gastric ulcer; H-Rg3, high-dose Rg3 group; L-Rg3, low-dose Rg3 group; M-Rg3, moderate-dose Rg3 group; SD, standard deviation.