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. 2019 Nov 1;19:275. doi: 10.1186/s12935-019-0958-z

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© The Author(s) 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 4 — UCA1 is capable of elevating PTP1B expression through inhibiting miR-206. a Bioinformatics prediction of the interaction between UCA1 and miR-206 through complementary base-pairs was shown. The mutant sequence of UCA1 is indicated. b Luciferase activities of pGL-PTP1B were analyzed by luciferase reporter gene assay in miR-206 and/or UCA1 (or UCA1-206-mut) transfected MCF-7 cells. c In miR-206 and/or UCA1 (or UCA1-206-mut) treated cells, PTP1B expression was assessed by Western blot. **p < 0.01 and no significance (NS), Student’s t-test