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. 2019 Oct 31;14:121. doi: 10.1186/s13000-019-0894-1

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© The Author(s). 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 1 — Establishment of a murine model bearing cardiac ischemia/reperfusion (I/R) injury. a Electrocardiogram monitoring of mice with (ischemia group) or without (Sham group) coronary artery occlusion. Mice after re-opening of the left anterior descending (LAD) artery (perfusion group) were also subject to ECG monitoring. b ELISA detection of serum cTnI levels in Sham group of mice and in mice with distinct perfusion time. c Hematoxylin & Eosin (HE) staining of heart tissues were performed. Myocardial cells in the sham group were well-ordered with regular structure, while myocardial cells from the I/R injured mice were swelling in the plasma (black arrow) and showed slight interstitial edema (blue arrow) or occasional cell hypertrophy (green arrow) after 3 or 6 h’ reperfusion. *, p < 0.05 vs. Sham