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. 2019 Nov 1;33(21-22):1475–1490. doi: 10.1101/gad.331397.119

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© 2019 Gaertner et al.; Published by Cold Spring Harbor Laboratory Press

This article is distributed exclusively by Cold Spring Harbor Laboratory Press for the first six months after the full-issue publication date (see http://genesdev.cshlp.org/site/misc/terms.xhtml). After six months, it is available under a Creative Commons License (Attribution-NonCommercial 4.0 International), as described at http://creativecommons.org/licenses/by-nc/4.0/.

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Figure 2. — Genome-scale next generation sequencing-based assays employed to study gene regulatory mechanisms of human pancreas differentiation. Schematic showing different genome-scale assays used to analyze chromatin remodeling, histone and DNA modifications, transcriptional regulation, gene expression, and expression of noncoding RNAs during pancreatic differentiation. (DNMTs) DNA methyltransferases, (TET) ten-eleven translocation enzymes, (HMTs) histone methlytransferases, (HATs) histone acetyltranferases, (TF) transcription factor, (RISC) RNA-induced silencing complex, (ATAC-seq) assay for transposase-accessible chromatin using sequencing, (WGBS) whole genome bisulfide sequencing, (ChIP-seq) chromatin immunoprecipitation sequencing.