Table 3.

Stamen-related features of JA-deficient mutants. Phospholipase A1 (PLA1); DEFECTIVE IN ANTHER DEHISCENCE1 (DAD1); 13-lipoxygenase (13-LOX); ALLENE OXIDE SYNTHASE (AOS); delayed dehiscence2-2 (dde2-2); 12-OXOPHYTODIENOIC ACID REDUCTASE3 (OPR3); delayed dehiscence1 (dde1).

Gene	Mutant	Phenotype	Species	Refs
PLA1/ DAD1	dad1	WT phenotype can be rescued by the JA application	Arabidopsis	[76]
		Developmental delay of flower bud opening
		Before flower opening, all cell types are normally developed in mutant anthers, similar to all structural features
		Pollen grains develop normally up to the trinucleate stage
		A defect in pollen grains occurs at the final stage of their maturation
		Defective in anther dehiscence
P0491E01		Normal anther development at the initial stages	rice	[81]
		Microspores development into mature pollen grains is impaired
LOX3 LOX4	lox3 lox4	Male sterile. JA application restored fertility	Arabidopsis	[82]
		Abnormal anther maturation
		Pollen is not viable
		Defective dehiscence
		Shorter filaments
AOS	dde2-2	Male-sterile phenotype which can be rescued by Me-JA application	Arabidopsis	[83]
		Impaired anther dehiscence and filament elongation
DDE1/ OPR3	dde1/opr3	WT phenotype can be rescued by the MeJA application	Arabidopsis	[84,85]
		Floral organs develop normally within the closed bud
		The anther locules do not dehisce at the time of flower opening
		Pollen develops to the trinucleate stage
		Pollen grains are predominantly inviable
		The filaments do not elongate sufficiently to position the locules above the stigma at anthesis