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. 2019 Oct 17;7(1):e000650. doi: 10.1136/bmjdrc-2019-000650

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© Author(s) (or their employer(s)) 2019. Re-use permitted under CC BY-NC. No commercial re-use. See rights and permissions. Published by BMJ.

This is an open access article distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited, appropriate credit is given, any changes made indicated, and the use is non-commercial. See: http://creativecommons.org/licenses/by-nc/4.0/.

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IL-1α deficiency completely inhibited HFD-induced accumulation of liver TGs. Liver specimens were obtained from male Loxp and IL-1α KO mice fed either regular chow or HFD (n=7–12 per group) for 16 weeks. (A) Total lipid weight in the liver normalized to liver weight. (B) Analysis of hepatic lipids with TLC. Data are presented as mean±SE. Asterisks/Hash marks depict statistically significant differences. *p≤0.05; **p≤0.01; ***p≤0.001 to Loxp. #p≤0.05; ##p≤0.01; ###p≤0.001 to chow (two-way ANOVA). ANOVA, analysis of variance; HFD, high-fat diet; IL, interleukin; KO, knockout; TG, triglyceride; TLC, thin-layer chromatography.