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. 2019 Nov 4;2:404. doi: 10.1038/s42003-019-0646-5

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — Wound-induced increase of H3K9/K14ac marking is associated with slower transcriptional activation. a Pie chart representing the percentage of genes associated with pre-wound H3K9/K14ac, post-wound H3K9/14ac and/or H3K4me3 among 3665 wound-induced genes. Genes are grouped based on their association with H3K9/14ac. b Spie chart representing the percentage of genes associated with pre-wound H3K9/K14ac, post-wound H3K9/14ac and/or post-wound H3K4me3 among genes within clusters 1–8. Genes are grouped based on their association with H3K9/14ac. The radii of the wedges correspond to the representation factor (hypergeometric test) of the epigenetic category in the cluster compared to its representation among all wound-induced genes. The black circle corresponds to a representation factor of 1 so that wedges inside the circle depict an underrepresented category and wedges that extend beyond the circle depict an overrepresented category. *p < 0.05, **p < 0.01, ***p < 0.001 (hypergeometric test)