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. 2018 Jul 3;24(5):285–296. doi: 10.1177/1753425918785008

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© The Author(s) 2018

Creative Commons Non Commercial CC BY-NC: This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 License (http://www.creativecommons.org/licenses/by-nc/4.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Figure 3. — LXA4 regulates LPS-induced inflammation via the ACE2-Ang-(1-7)-Mas axis in ALI. (a and b) The concentrations of TNF-α in the BALF (a) and serum (b). (c and d) The concentrations of IL-1β in the BALF (c) and serum (d). (e and f) The concentrations of IL-10 in the BALF (e) and serum (f). Data are presented as means ± SEM. *P < 0.05 and **P < 0.01 compared with control group, #P < 0.05 and ##P < 0.01 compared with LPS group, ΔP < 0.05 and ΔΔP < 0.01 compared with LPS + LXA4 group.