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. 2019 Nov 5;10:318. doi: 10.1186/s13287-019-1436-1

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© The Author(s). 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 6 — Depletion of TSG-6 from ASC-CCM fails to improve retinal GFAP expression in blast injury mice. GFAP immunoreactivity in the retina of a sham and b blast injury animals that received saline, c blast injury animals that received primed siControl ASC-CCM, and d blast injury animals that received siTSG-6 ASC-CCM. e ImageJ quantification of GFAP intensity. Data represent mean ± SEM from n = 5–9 animals/group. *p < 0.01; ***p < 0.001. Scale bar = 50 μm