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. 2019 Oct 16;11(10):2488. doi: 10.3390/nu11102488

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© 2019 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/).

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Effects of resveratrol on liver lipids, and also expression and phosphorylation of AMPK, ACC and Akt in liver tissue of GK and SD rats. Triglycerides (A), non-esterified fatty acids (B). Western blot immunodetection with antibodies to the indicated proteins, β-actin expression used as loading control (C). Protein levels relative to β-actin quantified by densitometry: Total AMPK (D) and pAMPK (E), total ACC (F) and pACC (G), total Akt (H) and pAkt (I). Results are representative of at least two independent experiments. GKC—Goto-Kakizaki control rats, GKR—Goto-Kakizaki resveratrol-treated rats, SDC—Sprague–Dawley control rats, SDR—Sprague–Dawley resveratrol-treated rats. Results are expressed as the means ± SEM from 10 rats in each group. Values with different letters are significantly different at p < 0.05.

Effects of resveratrol on liver lipids, and also expression and phosphorylation of AMPK, ACC and Akt in liver tissue of GK and SD rats. Triglycerides (A), non-esterified fatty acids (B). Western blot immunodetection with antibodies to the indicated proteins, β-actin expression used as loading control (C). Protein levels relative to β-actin quantified by densitometry: Total AMPK (D) and pAMPK (E), total ACC (F) and pACC (G), total Akt (H) and pAkt (I). Results are representative of at least two independent experiments. GKC—Goto-Kakizaki control rats, GKR—Goto-Kakizaki resveratrol-treated rats, SDC—Sprague–Dawley control rats, SDR—Sprague–Dawley resveratrol-treated rats. Results are expressed as the means ± SEM from 10 rats in each group. Values with different letters are significantly different at p < 0.05.