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. 2019 Mar 12;285(5):562–577. doi: 10.1111/joim.12877

Figure 4.

Figure 4

(a) Fluxes of apoB48 in CM, VLDL 1 and VLDL 2 and fluxes of apoB100 in VLDL 1 and VLDL 2; (b) Concentrations of apoB48 in CM, VLDL 1 and VLDL 2 and concentration of apoB100 in VLDL 1 and VLDL 2; (c) ApoB48‐TG flux in CM, VLDL 1 and VLDL 2 and apoB100‐TG flux in VLDL 1 and VLDL 2; (d) ApoB48‐TG concentration in CM, VLDL 1 and VLDL 2. Solid lines indicate model predictions and coloured circles indicate experimental data. Modelling of the previous day is indicated with grey background. The concentration and flux (in terms of mass) of apoB100 is higher than that of apoB48 in the VLDL 1/2 fractions. Total apoB48 flux into the CM fraction is higher than the basal apoB48 flux, and postprandial apoB48 flux also constitutes a significant portion of the total postprandial apoB48 flux. The total triglyceride flux into the CM fraction is the biggest source of triglyceride flux. However, VLDL 1‐TG concentration is higher than CM‐TG concentration because of the high CM‐TG FCR.