(A) Rad52∆ mutants have a constant high rate of GLMs but do not show a significant age-related increase in GLMs (p>0.05 cochran q test). Lifespan only shown 0–15 divisions due to reduced lifespan of rad52∆ mutants. (B) Survival curve showing the significant increase in GLM rates in individual rad52∆ mother cells. Each row is a separate mother cell, and the color indicates whether a cell experienced a normal cell cycle, GLM or terminal missegregation (n = 100 randomly selected cells). (C) Rad52∆ cells are more likely than wild-type to experience a terminal GLM, but any individual GLM is not more likely to result a terminal missegregation. This suggests Rad52 is important for preventing GLMs, but not for ensuring genomic content is properly segregated. (D) Cells lacking RAD52 have a statistically significant increase in the length of time a GLM lasts relative to wild-type cells (n is the number of cell cycles quantified, p<0.01 Students t-test).