FOXC2-AS1 |
DOX-induced cardiotoxicity in mice |
Down |
Inhibiting DOX-induced cardiomyocyte death [83] |
CHRF |
HF in mice under DOX treatment |
Up |
Promoting myocardial cell apoptosis [84] |
Mhrt |
Rat cardiomyopathy under DOX treatment |
Down |
Improving cell apoptosis of primary cardiomyocytes [85] |
MALAT1 |
IL-6 induced murine septic cardiomyocytes under LPS treatment |
Up |
Enhancing TNF-α expression in LPS-treated murine HL-1 cardiomyocytes [86] |
NEAT1 |
Sepsis in mice under LPS treatment |
Up |
Knockdown of NEAT1 could attenuate myocardial injury induced by LPS in mice [88] |
HOTAIR |
Sepsis in mice under LPS treatment |
Up |
Silencing of HOTAIR could preserve cardiac function in sepsis mice [89] |
Crnde |
DCM in mice |
Up |
Crnde could ameliorate cardiac fibrosis in DCM mice [92] |
MALAT1 |
DCM in rats |
Up |
MALAT1 knockdown could significantly reduce cardiomyocyte apoptosis and markedly improve left ventricular systolic and diastolic functions [93] |
|
DCM in mice and mice cardiomyocytes under high glucose treatment |
Up |
MALAT1 knockdown could reduce mice cardiomyocyte apoptosis induced by high glucose [94] |
H19 |
DCM in rats |
Down |
Inhibiting autophagy activation and improve cardiac function [95] |