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. 2019 Nov 12;8(1):1626–1635. doi: 10.1080/22221751.2019.1685913

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© 2019 The Author(s). Published by Informa UK Limited, trading as Taylor & Francis Group, on behalf of Shanghai Shangyixun Cultural Communication Co., Ltd

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 2. — Platelet-derived exosomes and microparticles interact with neutrophils dengue virus binds to platelet surface protein CLEC2 resulting in the release of microparticles and exosomes. The platelet releases microbodies then binds with neutrophil and macrophages resulting in the release of pro-inflammatory cytokines and components of neutrophil extracellular trap (NET) from these cells. Exosomes released from dengue virus-activated platelets also protect the bleeding in virus-infected patients.