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. 2019 Oct 23;20(6):4933–4942. doi: 10.3892/mmr.2019.10764

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Copyright: © Deng et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 1. — Exendin-4 facilitates bone defect repair. (A) Representative micro-computed tomography (µCT) scanning results of control mice bone (Con; n=6), defective bone (bd; n=6), defective bone receiving ADSCs (bd+ADSCs; n=6), and defective bone receiving ADSCs and exendin-4 (bd+ADSCs+Ex-4; n=6). (B) Quantitative presentation of microarchitectural parameters of control mice bone (Con; n=6), defective bone (bd; n=6), defective bone receiving ADSCs (bd+ADSCs; n=6), and defective bone receiving ADSCs and exendin-4 (bd+ADSCs+Ex-4; n=6). Error bars represent the mean ± SD. *P<0.05, **P<0.01 and ***P<0.001. (C) Calcein and tetracycline double staining of control mice bone (Con; n=6), defective bone (bd; n=6), defective bone receiving ADSCs (bd+ADSCs; n=6), and defective bone receiving ADSCs and exendin-4 (bd+ADSCs+Ex-4; n=6). ADSCs, adipose-derived stem cells.