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. 2019 Nov 8;10:1287. doi: 10.3389/fphar.2019.01287

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Copyright © 2019 Wu, Wang, Lin, Shang, Wang, Zhou, Wang, Zhang, Luo and Huang

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Molecular working model of CuC in cancer cells. (A) Western blot (WB) analysis of p-Akt and Akt in PC-3, T24, and HepG2 cells with or without CuC. p-Akt (Ser473) was significantly decreased in the presence of 100 nM CuC. Cells were treated for 24 h. (B) p-Akt (Ser473) and Akt expression in PC-3 and T24 cells under CuC (100 nM) treatment at different time points (0, 1, 3, 10, and 24 h). (C) Schematic diagram illustrated the bio-relevant context of antitumor activities of CuC, and the proposed cell growth arrest, apoptosis, and cell migration inhibition are illustrated.