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. 2019 Aug 23;74(12):3445–3452. doi: 10.1093/jac/dkz367

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© The Author(s) 2019. Published by Oxford University Press on behalf of the British Society for Antimicrobial Chemotherapy.

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/4.0/), which permits non-commercial re-use, distribution, and reproduction in any medium, provided the original work is properly cited. For commercial re-use, please contact journals.permissions@oup.com

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Figure 1. — Recombination-free phylogenetic tree including MLST, country, source, resistance, resistance gene and mutation data. Linezolid resistance in US isolates was attributed solely to the G2576T mutation of the 23S rRNA gene sequence. In contrast, linezolid resistance in Pakistan isolates resulted from different combinations of the acquired resistance genes optrA, poxtA and a cfr-like gene. Vancomycin resistance was observed in 90.6% (87/96) of the isolates and dalbavancin resistance was observed in 88.5% (85/96). Daptomycin resistance was observed in 3.13% (3/96) of the isolates with an additional 68.8% (66/96) classified as susceptible dose-dependent.