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. Author manuscript; available in PMC: 2020 Jul 1.
Published in final edited form as: Transl Res. 2019 Mar 27;209:14–21. doi: 10.1016/j.trsl.2019.03.006

Figure 2.

Figure 2.

Hypothetic model of mechanisms by which cellular senescence contributes to radiation-induced pulmonary fibrosis (RIPF). AEC2, type II alveolar epithelial cells; ASC, alveolar stem cells; ROS, reactive oxygen species SASP, senescence-associated secretary phenotype; RT, thoracic radiotherapy