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. 2019 Nov 16;16:227. doi: 10.1186/s12974-019-1597-y

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© The Author(s). 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 5 — Stat 3 phosphorylation after tMCAO. a Representative immunoblot results at 7 days after tMCAO. b Semi-quantitative analysis of p-Stat3 protein demonstrating that p-Stat3 expression was upregulated after tMCAO both in Ntg and GET-1 mice. p-Stat3 expression was robustly upregulated in the ipsilateral in GET-1 mice brain tissue than in Ntg mice 7 days after tMCAO. N = 3 per group pooled together and assayed thrice. *p < 0.05, ***p < 0.001; Contra = contralateral side of the ischemic brain; Ips = ipslateral side of the ischemic brain. c Representative images of p-Stat3 expression in ipsilateral SVZ of each group (red, indicated by white arrows). Scale bar = 100 μm; LV = lateral ventricle, SVZ = subventricular zone