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. Author manuscript; available in PMC: 2020 Sep 1.
Published in final edited form as: Pharmacol Ther. 2019 Apr 30;201:51–76. doi: 10.1016/j.pharmthera.2019.04.009

Table 4. NOP dysregulation in depression.

These data are compiled from preclinical and clinical studies that implicate NOP and N/OFQ in behavioral constructs that relate to depression under the domains of negative valence, positive valence, and arousal/regulatory systems. CeA – central nucleus of the amygdala, SNP – single nucleotide polymorphism, N/OFQ – nociceptin/orphaninFQ, NOP – nociceptin/orphaninFQ receptor, EPM – elevated plus maze, LDB – light /dark box, FST – forced swim test, LH – learned helplessness, LPS – lipopolysaccharide, SCN – suprachiasmatic nucleus.

Domain Constructs Behavioral effects Reference
Negative Valence: Acute threat (Fear) Systemic or intra-CeA administration of NOP agonists decreased freezing to the conditioned stimulus (Andero, et al., 2013; Witkin, et al., 2016)
G allele carriers of the rs6010719 SNP in the OPRL1 gene exhibited increased physiological startle measures of fear discrimination and greater functional connectivity between the amygdala and posterior insula. (Andero, et al., 2013)
Potential threat (Anxiety) N/OFQ enhanced thigmotaxis in the open field. (Fernandez, et al., 2004)
N/OFQ induced anxiogenic effects in rats on the EPM and LDB (Fernandez, et al., 2004)
NOP agonists produced anxiolytic effects (Duzzioni, et al., 2011)
NOP−/−mice exhibit reductions in anxiety like behavior compared to wildtype controls (Gavioli, et al., 2007)
Sustained threat (Aversive emotional state) NOP antagonists produce antidepressant-like effects in the FST, LH and LPS-induced depressive-like behavior (Asth, et al., 2016; Gavioli, et al., 2003; Gavioli, et al., 2004; Goeldner, et al., 2010; Holanda, et al., 2016; Medeiros, et al., 2015)
NOP−/−mice show reductions in depressive like behavior compared to their wildtype littermates. (Gavioli, et al., 2007)
Positive Valence: Reward Valuation NOP agonists stimulate feeding behavior (Ciccocioppo, et al., 2014; Nicholson, et al., 2002)
Arousal/Regulatory systems Circadian rhythm N/OFQ reduces neuronal activation in the SCN and can induce sedation (Gompf, et al., 2005)
NOP ligands were more efficacious when administered during the nadir of corticosterone secretion (Leggett, et al., 2007)