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. 2019 Oct 4;42(6):2371–2381. doi: 10.3892/or.2019.7354

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Copyright: © Lin et al.

This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, reproduction and adaptation in any medium and for any purpose provided that it is properly attributed. For attribution, the original author(s), title, publication source (PeerJ) and either DOI or URL of the article must be cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 1. — Expression of miR-183-5p.1 and TPM1 in GC cells and tissues. miR-183-5p.1 levels in (A) normal tissues, adjacent tissues, GC tissues and (B) normal gastric epithelial cells (GES-1) and GC cell lines (MKN-7, AGS, HGC-27) were determined by RT-qPCR. TPM1 mRNA expression in (C) normal tissues, adjacent tissues, GC tissues and (D) GES-1, MKN-7, AGS, HGC-27 cells as quantified by RT-qPCR. (E) TPM1 protein expression in GES-1 and AGS cells was determined by western blotting and (H) statistically analyzed. (F) TPM1 protein expression in GES-1 and MKN-7 cells was determined by western blotting and (I) statistically analyzed. (G) TPM1 protein expression in GES-1 and HGC-27 cells was determined by western blotting and (J) statistically analyzed. Results are presented as the mean ± standard error of the mean (n=3) *P<0.05, **P<0.01, ***P<0.001. GC, gastric cancer; RT-qPCR, reverse transcription-quantitative PCR; TPM1, tropomyosin 1; miR, microRNA.