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. 2019 Oct 4;42(6):2371–2381. doi: 10.3892/or.2019.7354

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Copyright: © Lin et al.

This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, reproduction and adaptation in any medium and for any purpose provided that it is properly attributed. For attribution, the original author(s), title, publication source (PeerJ) and either DOI or URL of the article must be cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 8. — Expression of miR-183-5p.1 and the apoptosis of GC AGS cells as analyzed by immunofluorescence. (A) The knockdown of miR-183-5p.1 promoted the apoptosis of AGS cells as determined by Hoechst 33258 and TUNEL assays. (B) Statistical results of apoptosis. (C) The overexpression of miR-183-5p.1 inhibited the apoptosis of AGS cells as determined by the Hoechst 33258 and TUNEL methods. (D) Statistical results of apoptosis. Results are presented as the mean ± standard error of the mean (n=3). **P<0.01. GC, gastric cancer; miR, microRNA.