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. 2019 Nov 15;18:1534735419886655. doi: 10.1177/1534735419886655

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© The Author(s) 2019

This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 License (http://www.creativecommons.org/licenses/by-nc/4.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Figure 1. — Roles of ginsenosides on function of tumor-associated macrpophages (TAMs) in tumor microenvironment.

In tumor microenvironment, TAMs inhibit the antitumor ability of T cells. Ginsenosides can block the inhibition function of TAM, promote TAMs conversion from M2 to M1 type, further inhibiting or killing tumor. PPT, 20(S)-Protopanaxatriol. Blue lines demonstrated the promotion () or inhibition () function among immune cells (TAM or T cells) and tumor cells. Yellow lines indicated the promotion () or inhibition () functions of ginsenosides.

Inline graphic — Roles of ginsenosides on function of tumor-associated macrpophages (TAMs) in tumor microenvironment.

In tumor microenvironment, TAMs inhibit the antitumor ability of T cells. Ginsenosides can block the inhibition function of TAM, promote TAMs conversion from M2 to M1 type, further inhibiting or killing tumor. PPT, 20(S)-Protopanaxatriol. Blue lines demonstrated the promotion () or inhibition () function among immune cells (TAM or T cells) and tumor cells. Yellow lines indicated the promotion () or inhibition () functions of ginsenosides.