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. 2019 Oct 14;18(6):4368–4378. doi: 10.3892/etm.2019.8102

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Copyright: © Cai et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 3. — Electron micrographs of Lp-infected guinea pig lung tissues 72 h after infection. (A) Ultrastructure in the lung tissues from the control group was normal. (B) In the Lp-infected group, the lung tissue was disordered. The surfaces of pulmonary capillary endothelial cells (indicated by arrow a) and alveolar epithelial cells (indicated by arrow b) were uneven, and the basement membrane between the two was blurred. (C) In the immunosuppressed group, the ultrastructure was close to normal. (D) In the lung tissues from the immunosuppressed Lp-infected group, destruction to the tissue structure was extensive and severe, with larger quantities of Lp infiltration (indicated by arrows). * denotes red blood cell and # denotes nucleus. Scale-bar, 2 µm. Lp, Legionella pneumophila.