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. 2019 Oct 25;18(6):4774–4780. doi: 10.3892/etm.2019.8132

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Copyright: © Chen et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 1. — Icariin rescues the EPC dysfunction induced by HG. (A) EPCs were treated with various concentrations of icariin for 24 h in HG conditions. Cell proliferation was determined by a Cell Counting Kit-8 assay. (B) EPCs were treated with or without 1 µM icariin in Transwell plates for 4 h. The migrated cells were counted under an optical microscope. (C) The relative levels of EPC migration. (D) EPCs cultured on a layer of Matrigel were treated with or without 1 µM icariin. The total length of the tube formation was measured in three random fields. for each group. (E) The relative levels of EPC tube formation. Data are presented as the mean ± SD of three individual experiments. *P<0.05 vs. Control group, ^#P<0.05 vs. HG group. EPC, endothelial progenitor cell; HG, high glucose.