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. 2019 Oct 25;18(6):4774–4780. doi: 10.3892/etm.2019.8132

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Copyright: © Chen et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 2. — Icariin inhibits the HG-induced phosphorylation of p38 and CREB in EPCs. (A) EPCs cultured in 5.5 (Control) or 25 mM glucose (HG) were treated with or without 1 µM icariin for 30 min. Levels of p-p38, total p38, p-CREB and total CREB were determined via western blot analysis. (B) Quantitative results of the phosphorylated levels of p38. (C) Quantitative results of the phosphorylated levels of CREB. Data are presented as the mean ± SD; n=3/group. *P<0.05 vs. Control group, ^#P<0.05 vs. HG group. CREB, cAMP response element binding protein; EPC, endothelial progenitor cell; HG, high glucose; p-, phosphorylated.