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. 2019 Oct 16;18(6):4329–4339. doi: 10.3892/etm.2019.8108

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Copyright: © Yousef et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 3. — PON1 and CK-MB levels in control, Fe₂O₃NP-exposed group, AgNP-exposed group, and Fe₂O₃NPs and AgNP-coexposed group. (A) Levels of PON1 in cardiac tissue and (B) in plasma. (C) Activity of CK-MB in cardiac tissues and (D) in plasma. *P<0.05 vs. control group; ^#P<0.05 vs. Fe₂O₃NPs exposed group; ^$P<0.05 vs. AgNPs exposed group; ^&P<0.05 synergistic interactions when both NPs are combined using Factorial Design. PON1, paraoxonase; CK-MB, creatine kinase-muscle/brain; Fe₂O₃NP, iron oxide nanoparticle; AgNP, silver nanoparticle.