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. 2019 Oct 28;24(21):3876. doi: 10.3390/molecules24213876

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© 2019 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/).

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Eleutheroside B attenuated programmed cell death induced by cisplatin in HK-2 cells. (A) Flow cytometry in HK-2 cells. (B) Western blotting analysis of programmed cell death-related molecules. (C) Immunofluorescence of phosphorylated-mixed lineage kinase domain like in HK-2 cells. Eleutheroside B significantly downregulated receptor-interacting protein kinase (RIPK)1, RIPK3, p-mixed lineage kinase domain-like protein (p-MLKL), and apoptosis-related protein cle-caspase3 in cisplatin-treated HK-2 cells. Data represent the mean ± SEM for 3–4 independent experiments in vitro. ** p < 0.01, *** p < 0.001 compared to normal; ^## p < 0.01 compared to model. Cis, cisplatin; EB, Eleutheroside B.