Table 4.

Differences between patients and controls in intrinsic functional connectivity with both STNs

	Pair of regions	Coordinates x/y/z (mm)	Mean Z controls	Mean Z patients	P	d′
Enhanced negative STN‐coupling in PD patients	L+R STN–L IPS/SPC/S1	−40/−48/56	0.019	−0.143	0.0001	−0.77
	L+R STN–R SPC	24/−64/62	0.008	−0.127	0.0003	−0.72
	L+R STN–R S1	34/−38/64	0.005	−0.144	0.0001	−0.78
	L+R STN–R IPS/SPC/S1	34/−48/50	0.012	−0.142	0.0001	−0.76
	L+R STN–L V1/V2	−4/−94/14	−0.004	−0.116	0.0011	−0.64
Attenuated negative STN‐coupling in PD patients	L+R STN–R Crus I	28/−76/−36	−0.113	0.007	0.0004	0.69
	L+R STN–R mid orbital/rectal gyrus	8/58/−10	−0.116	−0.013	0.0015	0.63

Mean Z _controls and Z _patients denote group‐averaged functional connectivity in 55 healthy participants and 54 patients with Parkinsons's disease (PD); d' denotes the effect size according to Cohen's definition [Cohen, 1988]. STN, subthalamic nucleus; SPC, superior parietal cortex; S1, primary sensory cortex; IPS, intraparietal sulcus; V1 and V2, subregions (area 17 and 18) of the visual cortex; L, left; R, right; Crus I is part of cerebellar lobule VIIa.