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. 2019 Nov 20;19:218. doi: 10.1186/s12890-019-0938-z

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© The Author(s). 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 8 — The effect of LY294002 on the activation of AKT induced by HDM and hrHsp90α in normal 16HBE cells. a:Western blotting analysis of the activation of AKT in normal 16HBE cells.(*P < 0.05 vs the control group,* ^#P < 0.05 vs the HDM group/the hrHsp90α group,n = 3). b:The transepithelial electrical resistance (TEER) was measured in normal 16HBE cells. Data shown are means±SD, (*P < 0.05 vs the control group,* ^#P < 0.05 vs the HDM group/the hrHsp90α group,n = 3). c:The transepithelial electrical resistance (TEER) was measured in normal 16HBE cells. Data shown are means±SD, (*P < 0.05 vs the control group,* ^#P < 0.05 vs the HDM group,n = 3)