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. 2019 Nov 20;10:335. doi: 10.1186/s13287-019-1410-y

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© The Author(s). 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 5 — Proangiogenic effects of DMOG-MSC-Exos in HUVECS after AKT/mTOR pathway was blocked. a DMOG-MSC-Exos were treated with MK2206 and blocking of AKT/mTOR signaling was detected by western blotting. b Wound healing assay in HUVECs treated with exosomes (50 μg/mL). c Tube-forming capacity of HUVECs treated with exosomes (50 μg/mL). d Quantitative analysis of wound closure. e Quantitative analysis of total tube length (*P < 0.05, versus control; #P < 0.05, versus MSC-Exos)