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. 2019 Nov 21;9:17276. doi: 10.1038/s41598-019-53660-x

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Venn diagram depicting both shared and exclusive pathogenic variants in CTC subsets. Numbers of the variants shared between the subsets are differently colored while those of the exclusive mutations are highlighted in black (left). Shared variants involved only 4 genes (ATM, FGFR3, TP53, and PIK3CA), whereas the subset-specific mutations were at higher number of genes in each CTC subpopulation (right).