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. 2019 Nov 22;9:17369. doi: 10.1038/s41598-019-53910-y

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Inhibition of CDK9 attenuated pro-inflammatory genes in monocytes following P. gingivalis infection. (A–D) THP-1 monocytic cells were preincubated with FVD to block CDK9 for 2 hours and then infected with P. gingivalis. Production of cytokine were assessed by ELISA at 4 and 24 hours after infection. (E–H) PBMCs were pretreated with FVD and then infected with P. gingivalis. Cytokine production in the culture was detected by ELISA. (**p < 0.01; ***p < 0.001).