TABLE 1.
Demographic and imaging features of the study participants.
| ID | Lesion | Goldmann VF perimetry (Y/N) | Goldmann VF perimetry findings | |||
| Epilepsy/Tumor | Side | Description and Location (Optic radiation component involved) | Pathology | |||
| 1 | Tumor | L | Expanding solitary thalamic lesion (LGB) | Pilocytic astrocytoma | N | Intact (on confrontation) |
| 2 | Tumor | R | Previous R anterior temporal lesionectomy (ML) | Recurrent anaplastic ependymoma | Y | Left HSQ-VFD∗ |
| 3 | Epilepsy | R | Small, deformed thalamus. Extensive frontal, temporal and parietal cortical thickening and SHE; Exca vaco ventricular dilatation (entire OR) | Hemispheric FCD sparing occipital lobe | N | Intact (on confrontation) |
| 4 | Epilepsy | L | Expanding angular gyrus lesion, displacing and infiltrating into the SS (Principally PB in the SS) | Protoplasmic astrocytoma | Y | Intact |
| Mid | Midline pineal lesion (LGB) | Pineal region colloid cyst | ||||
| 5 | Epilepsy | R | Gliosis of the fusiform and inferior temporal gyrus (Principally ML in the SS) | FCD and gliosis secondary to neonatal cerebral hemorrhage | Y | Intact |
| 6 | Epilepsy | L | Expanding lingual gyrus lesion, displacing the inferior calcarine cortex (ML and MB) | Pleomorphic Xanthoastrocytoma | Y | Intact |
| 7 | Epilepsy | R | Previous occipital lobectomy with minimal residual occipital lobe showing atrophic changes (entire OR; missing PCCx) | FCD and gliosis due to neonatal ischemic vascular injury | Y | Left HH-VFD∗ |
| 8 | Epilepsy | R | Anterior temporal gliosis; previous tumor resection (ML) | Gliosis | Y | Left HSQ-VFD∗ |
FCD, focal cortical dysplasia; HH, homonymous hemianopsia; HSQ, homonymous superior quadranopsia; L, left; MB, middle bundle; ML, Meyer’s loop (anterior bundle); Mid, midline; N, no; PCCx, peri-calcarine cortex; PB, posterior bundle; VFD, visual field deficits; R, right; SHE, subependymal heterotopia; SS, sagittal stratum; Y, yes. ∗Pre-existing visual field deficits from previous epilepsy surgeries.