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. 2019 Nov 26;9:17621. doi: 10.1038/s41598-019-54098-x

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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mDIO alters transcript levels of key enzymes and transcription factors that regulate fetal hepatic gluconeogenesis. (A) Relative mRNA levels of; peroxisome proliferator-activated receptor gamma 1 alpha (Pgc1α), pyruvate carboxylase (Pc), glucose-6-phosphate (G6p), hepatocyte nuclear factor 4 alpha (Hnf4α), phosphoenolpyruvate carboxylase (pepck) in the liver of control and high-fat exposed fetuses. (B) Relative mRNA levels of liver X receptor alpha (Nr1h3) and liver X receptor beta (Nr1h2) in the liver of control and high-fat exposed fetuses. Data are presented as box and whisker plots, min to max, where the centre line represents the median. Data were analyzed using a Student’s t-test. Control: open boxes, (n = 7) and high-fat: grey boxes, (n = 7) *p < 0.05.