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. 2019 Nov 27;19:313. doi: 10.1186/s12935-019-0968-x

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© The Author(s) 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 5 — Knockdown of DLX6-AS1 inhibits tumor growth in vivo. a Relative expression levels of DLX6-AS1 in SK‐N‐SH cells stably transfected with shNC or DLX6-AS1 by qRT‐PCR. b Tumor volume was measured in shNC group and shDLX6-AS1 group at indicated times. c The weight of dissected tumors in shDLX6-AS1 group was lower than that in shNC group. The expression of d DLX6-AS1 and e miR-107 in dissected tumor tissues from shNC group and shDLX6-AS1 group were determined by qRT-PCR. f The BDNF, GAP43 and NF-200 protein expression of dissected tumor tissues were determined by western blot. g Survival analysis showing the overall survival of mice treated with shNC or shDLX6-AS1 SH-SY5Y cells. **p < 0.01 and ***p < 0.001