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. 2019 Nov 27;9:17727. doi: 10.1038/s41598-019-54177-z

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Sema4C mediates the functional effects of miR-31-3p and EMT in CC cells. (A,C) The repressive effects of miR-31-3p on EMT were rescued by overexpression of Sema4C. (B,D–F) Sema4C restoration abrogated the inhibitory effects on migration and invasion of Caski cells induced by miR-31-3p. Data are presented as mean ± SD. P value was analyzed by paired student’s t test, ^#P = 0.019 for cell migration and ^#P = 0.014 for cell invasion, n = 3.