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. 2019 Nov 29;9(12):96. doi: 10.1038/s41408-019-0257-x

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — A total number of 183 BCP-ALL cases were analyzed with the gc-HTS approach. Nineteen of them had a known BCR-ABL1 fusion identified by either FISH or molecular genetics. The 164 remaining cases were further divided in three subgroups according to the MRD status at EOI therapy. IKZF1 deletions/mutations were detected by MLPA or Sanger sequencing. Genomic fusion breakpoints were used for quantification of follow-up samples by RQ-PCR.