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. 2019 Nov 29;9:17897. doi: 10.1038/s41598-019-52872-5

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Ischemic-hypoxia elicits podocyte injury. (A) Staining for WT1 in sham and MCAO rat glomerular sections. The scale bar represents images of 10 µm and images were captured with a 100x objective of Leica trinocular microscope. (B) TEM images of podocyte foot processes in sham and MCAO rat kidney sections. In ischemic-stroke rats, podocyte foot-processes were small and the thickness of GBM was increased compared to sham-operated rats.